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  1. CRISPR activation for SCN2A-related neurodevelopmental disorders.

    Publication

  2. Cellular and behavioral effects of altered NaV1.2 sodium channel ion permeability in Scn2aK1422E mice.

    Publication

  3. Physical and functional convergence of the autism risk genes Scn2a and Ank2 in neocortical pyramidal cell dendrites.

    Publication

  4. CRISPR activation rescues abnormalities in SCN2A haploinsufficiency-associated autism spectrum disorder.

    Publication

  5. LiCl treatment leads to long-term restoration of spine maturation and synaptogenesis in adult Tbr1 mutants.

    Publication

  6. Dendritic integration dysfunction in neurodevelopmental disorders.

    Publication

  7. Developmental dynamics of voltage-gated sodium channel isoform expression in the human and mouse neocortex.

    Publication

  8. Paradoxical hyperexcitability from NAV1.2 sodium channel loss in neocortical pyramidal cells.

    Publication

  9. Soma-targeted imaging of neural circuits by ribosome tethering.

    Publication

  10. Alternative splicing potentiates dysfunction of early-onset epileptic encephalopathy SCN2A variants.

    Publication

  11. The autism-associated gene Scn2a contributes to dendritic excitability and synaptic function in the prefrontal cortex.

    Publication

  12. Progress in understanding and treating SCN2A-mediated disorders.

    Publication

  13. Opposing effects on Nav1.2 function underlie differences between SCN2A variants observed in individuals with autism spectrum disorder or infantile seizures.

    Publication