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  1. Polygenic and developmental profiles of autism differ by age at diagnosis.

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  2. Phenotypic effects of genetic variants associated with autism.

    Publication

  3. Processing of social and monetary rewards in autism spectrum disorders.

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  4. Tackling hypo and hyper sensory processing heterogeneity in autism: From clinical stratification to genetic pathways.

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  5. Genetic correlates of phenotypic heterogeneity in autism.

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  6. Genome-wide analyses of individual differences in quantitatively assessed reading- and language-related skills in up to 34,000 people.

    Publication

  7. Resting state EEG power spectrum and functional connectivity in autism: A cross-sectional analysis.

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  8. Fine-grained topographic organization within somatosensory cortex during resting-state and emotional face-matching task and its association with ASD traits.

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  9. The link between autism and sex-specific neuroanatomy, and associated cognition and gene expression.

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  10. Excitatory/inhibitory imbalance in autism: The role of glutamate and GABA gene-sets in symptoms and cortical brain structure.

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  11. Preference for biological motion is reduced in ASD: Implications for clinical trials and the search for biomarkers.

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  12. The meaning of significant mean group differences for biomarker discovery.

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  13. A recurrent SHANK3 frameshift variant in autism spectrum disorder.

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  14. Genome-wide analysis of gene dosage in 24,092 individuals estimates that 10,000 genes modulate cognitive ability.

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  15. Atlas of functional connectivity relationships across rare and common genetic variants, traits, and psychiatric conditions.

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  16. Imbalanced social-communicative and restricted repetitive behavior subtypes of autism spectrum disorder exhibit different neural circuitry.

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  17. Effect sizes of deletions and duplications on autism risk across the genome.

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  18. Fractionating autism based on neuroanatomical normative modeling.

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  19. Mutations associated with neuropsychiatric conditions delineate functional brain connectivity dimensions contributing to autism and schizophrenia.

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  20. Insufficient evidence for “autism-specific” genes.

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  21. Social and non-social autism symptoms and trait domains are genetically dissociable.

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  22. Both rare and common genetic variants contribute to autism in the Faroe Islands.

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  23. Disruption of melatonin synthesis is associated with impaired 14-3-3 and miR-451 levels in patients with autism spectrum disorders.

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  24. CNTNAP4 differentially contributes to GABAergic and dopaminergic synaptic transmission.

    Publication